Precard (Prekard)

Has antihypoxic effect.

Directly affecting the cardiomyocytes and neurons of the brain, it optimizes their metabolism and function. The cytoprotective effect is due to an increase in the energy potential, activation of oxidative decarboxylation and rationalization of oxygen consumption (enhancement of aerobic glycolysis and fatty acid oxidation blockade due to inhibition of long chain 3-ketoatsil-co-thiolase). Supports contractility of the myocardium, prevents intracellular depletion of adenosine triphosphate (ATP) and creatine phosphate. In conditions of acidosis normalizes the functioning of ion channels of membranes, prevents the accumulation of Ca2 + and Na + in cardiomyocytes, normalizes the intracellular concentration of K +.

Reduces intracellularityacidosis and phosphorus concentrationatheroma caused by myocardial ischemia and reperfusion. Prevents the damaging effect of free radicals, preserves the integrity of cell membranes, prevents the activation of neutrophils in the ischemic zone, increases the duration of electricityreduces the potential for yield of creatine phosphokinase (CK) from cells and severity of ischemic myocardial damage.

When angina pectoris reduces the frequency of attacks (decreases the need for nitrates), after 2 weeks of treatment, exercise tolerance increases, sharp fluctuations in blood pressure decrease. Reduces dizziness and noise in the ears of ischemic etiology. In vascular pathology, the eye restores the functional activity of the retina.